Linking pathological narcissism to posttraumatic stress disorder in Veterans

Abstract: Combat deployments are stressful life events that confer risk for posttraumatic stress disorder (PTSD) in veterans. Typically, PTSD is conceptualized as being caused by traumatic experiences, leading research to focus predominantly on events while excluding other contributors, such as personality features. Narcissism may contribute to the deleterious effects of trauma exposure because of vulnerabilities in the sense of self, as demonstrated in research on narcissism and the development and maintenance of PTSD in civilians. However, the strength of the association between pathological narcissism features and PTSD has yet to be examined in a veteran sample. The present study sought to address this gap by comparing how narcissism contributes to variance in PTSD symptoms relative to the contribution of combat experience. In a sample of veterans deployed in support of recent operations in Afghanistan and Iraq (N = 179), regression analysis showed that higher pathological narcissism features accounted for variance beyond combat experience alone in PTSD symptoms, ∆R2 = .13, p < .001. When dimensions of narcissism were examined as separate predictors of PTSD, vulnerable, β = .45, p < .001, but not grandiose, β = −.09, p = .293, features had a significant effect on PTSD. Our results align with recent work demonstrating that personality pathology is an important factor in the study of PTSD in veterans. Further research incorporating a larger variety of variables related to personality functioning, personality traits, and life events is needed to understand the role of pathological narcissism features in the development of PTSD.

Read the full article
Report a problem with this article

Related articles

  • More for Researchers

    Risk of incident mild cognitive impairment and dementia soon after leaving incarceration among a US Veteran population

    Abstract: Objectives: Increasing numbers of older adults are reentering community following incarceration (i.e., reentry), yet risk of incident neurodegenerative disorders associated with reentry is unknown. Our objective was to determine association between reentry status (reentry vs never-incarcerated) and mild cognitive impairment (MCI) and/or dementia. Methods: This nationwide, longitudinal cohort study used linked Centers for Medicare & Medicaid Services and Veterans Health Administration data. Participants were aged 65 years or older who experienced reentry between October 1, 2012, and December 31, 2018, with no preincarceration MCI/dementia, compared with age-matched/sex-matched never-incarcerated veterans. MCI/dementia was defined by diagnostic codes. Fine-Gray proportional hazards models were used to examine association. Results: This study included 35,520 veterans, mean age of 70 years, and approximately 1% women. The reentry group (N = 5,920) had higher incidence of MCI/dementia compared with the never-incarcerated group (N = 29,600; 10.2% vs 7.2%; fully adjusted hazard ratio [aHR] 1.12; 95% CI 1.00-1.25). On further investigation, reentry was associated with increased risk of dementia with or without prior MCI diagnosis (aHR 1.21; 95% CI 1.06-1.39) but not MCI only. Discussion: Transition from incarceration to community increased risk of neurocognitive diagnosis. Findings indicate health/social services to identify and address significant cognitive deficits on late-life reentry. Limitations include generalizability to nonveterans.